Effects of infant formula on cell homeostasis and cytokine levels in an in vivo and in vitro murine aspiration model

Pediatr Pulmonol. 2011 Sep;46(9):927-33. doi: 10.1002/ppul.21444. Epub 2011 Apr 25.

Abstract

Rationale: The role of infant formula aspiration in lung injury has not been studied extensively. We evaluated the effects of a single infant formula aspiration into the lungs of mice and the effect of infant formula exposure on cell lines representing murine alveolar macrophages and type II epithelial cells.

Objectives: To study the effects of exposure to infant formula on cell count histology and cytokine levels in an in vivo and in vitro model of aspiration.

Methods: In vivo: Juvenile mice received 2.5 µl/g of 50% infant formula intranasally. Bronchoalveolar lavage samples were collected at 1, 2, and 7 days after aspiration and evaluated for cell count and differential. In vitro: RAW 264.7 and MLE-15 cells were exposed to 1% infant formula for 6 hr. Extracellular levels of IL-6, TNF-α, MIP-2, and KC were measured in lavage fluid and cell media using ELISA assays.

Results: In vivo: An increase in neutrophils, IL-6 and KC levels were noted 24 hr after infant formula exposure. In vitro: An increase in TNF-α levels from RAW 264.7 and MIP-2 and KC levels from MLE-15 cells was noted after infant formula exposure.

Conclusions: A single aspiration of infant formula into the lungs leads to an acute inflammatory response involving both lung macrophages and epithelial cells.

MeSH terms

  • Animals
  • Bronchoalveolar Lavage Fluid / chemistry
  • Bronchoalveolar Lavage Fluid / cytology
  • Cell Line
  • Cytokines / biosynthesis*
  • Disease Models, Animal
  • Homeostasis / drug effects*
  • Humans
  • Infant
  • Infant Formula / pharmacology*
  • Inflammation / chemically induced
  • Inflammation / metabolism
  • Lung / drug effects
  • Lung / metabolism
  • Macrophages, Alveolar / drug effects
  • Macrophages, Alveolar / metabolism
  • Mice
  • Mice, Inbred BALB C
  • Neutrophils / drug effects
  • Neutrophils / metabolism
  • Pneumonia, Aspiration / metabolism*

Substances

  • Cytokines