Feasibility of a novel positive feedback effect of 131I-promoted Bac-Egr1-hNIS expression in malignant glioma via baculovirus

Nucl Med Biol. 2011 May;38(4):599-604. doi: 10.1016/j.nucmedbio.2010.11.005. Epub 2011 Feb 4.

Abstract

Purpose: As intracellular iodine is released rapidly, increased expression of sodium/iodide symporter (NIS) is required for effective radioiodine treatment of tumor. As Egr1 promoter is activated by ¹³¹I and may promote human NIS (hNIS) expression, hNIS also induces ¹³¹I uptake and activates Egr1, so the existence of a positive feedback effect of ¹³¹I-promoted Egr1-hNIS expression is possible. Our purpose was to investigate the possible existence of this positive feedback effect through a series of in vitro pioneer studies.

Method: Recombinant baculovirus (Bac-Egr1-hNIS) encoding the hNIS gene under the control of a radiation-inducible Egrl promoter was constructed. To test ¹³¹I-promoted hNIS expression, human malignant glioma U87 cells were transfected with Bac-Egr1-hNIS, stimulated with or without ¹³¹I; the expression of hNIS protein was detected by immunofluorescence and flow cytometry test. In addition, the uptake and efflux of ¹³¹I were determined after the incubation of Bac-Egr1-hNIS-transfected U87 cells with or without ¹³¹I.

Results: Immunocytochemical staining and flow cytometry test showed a higher hNIS protein expression in Bac-Egr1-hNIS-transfected U87 cells with ¹³¹I stimulation than in cells without stimulation. Bac-Egr1-hNIS-transfected U87 cells accumulated up to about 4.05 times of ¹³¹I after ¹³¹I stimulation. The amount of ¹³¹I uptake in both groups showed a baculovirus dose-dependent manner. However, rapid efflux of radioactivity was observed in both groups, with 50% lost during the first 2 min after the ¹³¹I-containing medium had been replaced by a nonradioactive medium.

Conclusion: Our results indicated that an improved transgene expression of ¹³¹I-stimulated hNIS in U87 cells using a baculovirus vector containing the Egr1 promoter is possible, and the increased expression of hNIS is responsible for a higher ¹³¹I uptake. It might provide a reference for the existence of a positive feedback effect in ¹³¹I-promoted Bac-Egr1-hNIS expression in malignant glioma and is an interesting aspect of NIS-related studies.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Baculoviridae / genetics*
  • Biological Transport
  • Cell Line, Tumor
  • Dose-Response Relationship, Drug
  • Early Growth Response Protein 1 / genetics*
  • Feasibility Studies
  • Feedback, Physiological / drug effects*
  • Gene Expression / drug effects
  • Genetic Vectors / genetics
  • Glioma / genetics*
  • Glioma / metabolism
  • Glioma / physiopathology
  • Humans
  • Iodine Radioisotopes / metabolism
  • Iodine Radioisotopes / pharmacology
  • Promoter Regions, Genetic / genetics
  • Symporters / genetics*
  • Transfection
  • Transgenes / genetics

Substances

  • Early Growth Response Protein 1
  • Iodine Radioisotopes
  • Symporters
  • sodium-iodide symporter