A case of acquired Fanconi syndrome induced by zoledronic acid

Intern Med. 2011;50(9):1075-9. doi: 10.2169/internalmedicine.50.4855. Epub 2011 May 1.

Abstract

A 61-year-old woman with metastatic breast cancer was diagnosed as having acquired Fanconi syndrome. In this case, the cause of this syndrome was most likely zoledronic acid (Zometa), which had been infused intravenously at a dose of 4 mg over 15 minutes weekly because of malignancy-associated hypercalcemia. Zoledronic acid is nephrotoxic and may induce severe tubular dysfunction, which can cause development of Fanconi syndrome. Therefore, close monitoring of proximal tubular function is recommended during therapy with zoledronic acid, especially when frequent administration of zoledronic acid is needed.

Publication types

  • Case Reports

MeSH terms

  • Antineoplastic Combined Chemotherapy Protocols / therapeutic use
  • Bone Density Conservation Agents / adverse effects*
  • Breast Neoplasms / complications
  • Breast Neoplasms / drug therapy
  • Breast Neoplasms / secondary
  • Diphosphonates / adverse effects*
  • Fanconi Syndrome / chemically induced*
  • Fanconi Syndrome / diagnosis
  • Fanconi Syndrome / urine
  • Female
  • Humans
  • Hypercalcemia / drug therapy
  • Hypercalcemia / etiology
  • Imidazoles / adverse effects*
  • Kidney Function Tests
  • Middle Aged
  • Zoledronic Acid

Substances

  • Bone Density Conservation Agents
  • Diphosphonates
  • Imidazoles
  • Zoledronic Acid