Gene expression profiles in BCL11B-siRNA treated malignant T cells

J Hematol Oncol. 2011 May 15:4:23. doi: 10.1186/1756-8722-4-23.

Abstract

Background: Downregulation of the B-cell chronic lymphocytic leukemia (CLL)/lymphoma11B (BCL11B) gene by small interfering RNA (siRNA) leads to growth inhibition and apoptosis of the human T-cell acute lymphoblastic leukemia (T-ALL) cell line Molt-4. To further characterize the molecular mechanism, a global gene expression profile of BCL11B-siRNA -treated Molt-4 cells was established. The expression profiles of several genes were further validated in the BCL11B-siRNA -treated Molt-4 cells and primary T-ALL cells.

Results: 142 genes were found to be upregulated and 109 genes downregulated in the BCL11B-siRNA -treated Molt-4 cells by microarray analysis. Among apoptosis-related genes, three pro-apoptotic genes, TNFSF10, BIK, BNIP3, were upregulated and one anti-apoptotic gene, BCL2L1 was downregulated. Moreover, the expression of SPP1 and CREBBP genes involved in the transforming growth factor (TGF-β) pathway was down 16-fold. Expression levels of TNFSF10, BCL2L1, SPP1, and CREBBP were also examined by real-time PCR. A similar expression pattern of TNFSF10, BCL2L1, and SPP1 was identified. However, CREBBP was not downregulated in the BLC11B-siRNA -treated Molt-4 cells.

Conclusion: BCL11B-siRNA treatment altered expression profiles of TNFSF10, BCL2L1, and SPP1 in both Molt-4 T cell line and primary T-ALL cells.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • CREB-Binding Protein / biosynthesis
  • CREB-Binding Protein / genetics
  • Cell Line, Tumor
  • Child
  • Down-Regulation
  • Female
  • Gene Expression Profiling
  • Gene Expression Regulation, Leukemic*
  • Humans
  • Leukemia, Lymphocytic, Chronic, B-Cell / genetics
  • Leukemia, Lymphocytic, Chronic, B-Cell / metabolism
  • Male
  • Middle Aged
  • Precursor T-Cell Lymphoblastic Leukemia-Lymphoma / genetics*
  • Precursor T-Cell Lymphoblastic Leukemia-Lymphoma / metabolism
  • RNA, Small Interfering / administration & dosage
  • RNA, Small Interfering / genetics*
  • Repressor Proteins / genetics*
  • TNF-Related Apoptosis-Inducing Ligand / biosynthesis
  • TNF-Related Apoptosis-Inducing Ligand / genetics
  • Tumor Suppressor Proteins / genetics*
  • Up-Regulation
  • bcl-X Protein / biosynthesis
  • bcl-X Protein / genetics

Substances

  • BCL11B protein, human
  • BCL2L1 protein, human
  • RNA, Small Interfering
  • Repressor Proteins
  • TNF-Related Apoptosis-Inducing Ligand
  • TNFSF10 protein, human
  • Tumor Suppressor Proteins
  • bcl-X Protein
  • CREB-Binding Protein
  • CREBBP protein, human