[Kidney injury after cardiopulmonary bypass in infants with congenital heart disease]

Zhongguo Dang Dai Er Ke Za Zhi. 2011 May;13(5):385-7.
[Article in Chinese]

Abstract

Objective: To study kidney injury in infants with congenital heart disease (CHD) who underwent cardiac surgery with cardiopulmonary bypass (CPB).

Methods: Forty CHD infants undergoing cardiac surgery with CPB from October 2009 to July 2010 were enrolled. The concentrations of serum tumor necrosis factor-alpha (TNF-α), interleukin-6 (IL-6), cystatin C (CysC) and urinary N-acetyl-beta-D-glucosaminidase (NAG) were detected using ELISA before bypass, at the end of surgery, and 2 hrs, 6 hrs and 24 hrs after surgery. Serum concentrations of creatinine (Cr) and urea nitrogen (BUN) were measured with conventional biochemistry technique before and after surgery.

Results: The concentrations of serum Cr and BUN were normal before and after surgery. After CPB, the concentrations of serum TNF-α and IL-6 and urinary NAG increased significantly (P<0.05). Serum TNF-α was positively correlated with urinary NAG and serum CysC (r=0.195, 0.190, respectively; both P<0.05). Serum IL-6 was positively correlated with urinary NAG (r=0.278, P<0.01). The positive rate in kidney injury was detected by serum CysC and urinary NAG were significantly higher than by serum Cr or BUN (both P<0.01).

Conclusions: CPB can cause acute kidney injury in infants, which may be correlated with the increase in the concentrations of serum TNF-α and IL-6. Serum CysC and urinary NAG may be used as sensitive markers for reflecting the changes of renal function.

Publication types

  • English Abstract

MeSH terms

  • Acetylglucosaminidase / urine
  • Acute Kidney Injury / etiology*
  • Cardiopulmonary Bypass / adverse effects*
  • Child
  • Child, Preschool
  • Cystatin C / blood
  • Female
  • Heart Defects, Congenital / surgery*
  • Humans
  • Interleukin-6 / blood
  • Male
  • Tumor Necrosis Factor-alpha / blood

Substances

  • Cystatin C
  • Interleukin-6
  • Tumor Necrosis Factor-alpha
  • Acetylglucosaminidase