Notch destabilises maternal beta-catenin and restricts dorsal-anterior development in Xenopus

Development. 2011 Jun;138(12):2567-79. doi: 10.1242/dev.061143.

Abstract

The blastula chordin- and noggin-expressing centre (BCNE) is the predecessor of the Spemann-Mangold's organiser and also contains the precursors of the brain. This signalling centre comprises animal-dorsal and marginal-dorsal cells and appears as a consequence of the nuclear accumulation of β-catenin on the dorsal side. Here, we propose a role for Notch that was not previously explored during early development in vertebrates. Notch initially destabilises β-catenin in a process that does not depend on its phosphorylation by GSK3. This is important to restrict the BCNE to its normal extent and to control the size of the brain.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Brain / embryology*
  • Mothers
  • Organ Size
  • Protein Stability
  • Receptors, Notch / physiology*
  • Xenopus Proteins / metabolism*
  • Xenopus laevis / embryology*
  • beta Catenin / metabolism*

Substances

  • CTNNB1 protein, Xenopus
  • NOTCH1 protein, Xenopus
  • Receptors, Notch
  • Xenopus Proteins
  • beta Catenin