Endothelium-derived hyperpolarizing factor in the brain: influence of sex, vessel size and disease state

Womens Health (Lond). 2011 May;7(3):293-303. doi: 10.2217/whe.11.26.

Abstract

The endothelial layer of cells lining the intimal surface of blood vessels is essential for vascular function. The endothelium releases multiple vasodilator and protective factors, including nitric oxide, prostacyclin and endothelium-derived hyperpolarizing factor; an imbalance in these factors predisposes individuals to vascular diseases such as stroke. These factors are differentially regulated by vessel size, sex hormones and disease state, therefore playing differential roles in different tissues following vascular injury. In particular, the endothelium-derived hyperpolarizing factor candidate termed epoxyeicosatrienoic acid, plays a prominent role in microvessel function, especially after ischemia, thereby making this signaling pathway an attractive target for therapy in vascular disease, including stroke.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Biological Factors* / physiology
  • Brain / physiopathology
  • Endothelium, Vascular / physiopathology
  • Epoxide Hydrolases / genetics
  • Female
  • Humans
  • Male
  • Nitric Oxide / metabolism
  • Sex Factors
  • Stroke / etiology*
  • Stroke / physiopathology*
  • Stroke / therapy
  • Vasodilation

Substances

  • Biological Factors
  • endothelium-dependent hyperpolarization factor
  • Nitric Oxide
  • Epoxide Hydrolases
  • EPHX2 protein, human