p16INK4a methylation in serum as a follow-up marker for recurrence of colorectal cancer

Anticancer Res. 2011 May;31(5):1643-6.

Abstract

Background: p16(INK4a) methylation present in the tumors of colorectal cancer (CRC) patients can be detected in their serum using quantitative methylation-specific PCR (Q-MSP). To investigate the possibility that this technique could be applied to the monitoring for cancer recurrence in CRC patients, p16(INK4a) methylation in the serum of CRC patients during their follow-up period was evaluated.

Materials and methods: Using Q-MSP on serum samples from 21 CRC patients undergoing surgery for primary CRC, the p16INK4a methylation score (p16(INK4a) MS) was evaluated one day before surgery and during the follow-up period.

Results: In the serum samples collected before primary resection, p16(INK4a) methylation was detected in 8 out of the 13 patients with same methylation in the tumor. The p16(INK4a) MS decreased within 2 weeks after surgery. Only two patients, who had the potential for recurrence, exhibited p16(INK4a) methylation in their serum. One month after surgery, in the patients with recurrence of tumor, a dramatic increase in p16(INK4a) MS was observed, while in the disease-free patients no methylation was seen continuously.

Conclusion: p16(INK4a) MS could sensitively reflect the recurrence status and may be useful for identifying the presence of recurrence during the follow-up of CRC patients.

Publication types

  • Comparative Study

MeSH terms

  • Aged
  • Biomarkers, Tumor / blood
  • Biomarkers, Tumor / genetics*
  • Case-Control Studies
  • Colorectal Neoplasms / blood
  • Colorectal Neoplasms / genetics*
  • Colorectal Neoplasms / pathology
  • Colorectal Neoplasms / surgery
  • Cyclin-Dependent Kinase Inhibitor p16 / blood*
  • Cyclin-Dependent Kinase Inhibitor p16 / genetics*
  • DNA Methylation*
  • DNA, Neoplasm / genetics
  • Female
  • Follow-Up Studies
  • Humans
  • Lymphatic Metastasis
  • Male
  • Neoplasm Recurrence, Local / blood
  • Neoplasm Recurrence, Local / diagnosis*
  • Neoplasm Recurrence, Local / genetics*
  • Neoplasm Recurrence, Local / surgery
  • Polymerase Chain Reaction
  • Prognosis
  • Promoter Regions, Genetic

Substances

  • Biomarkers, Tumor
  • Cyclin-Dependent Kinase Inhibitor p16
  • DNA, Neoplasm