Abstract
The histone 3 lysine 79 (H3K79) methyltransferase Dot1l has been implicated in the development of leukemias bearing translocations of the Mixed Lineage Leukemia (MLL) gene. We identified the MLL-fusion targets in an MLL-AF9 leukemia model, and conducted epigenetic profiling for H3K79me2, H3K4me3, H3K27me3, and H3K36me3 in hematopoietic progenitor and leukemia stem cells (LSCs). We found abnormal profiles only for H3K79me2 on MLL-AF9 fusion target loci in LSCs. Inactivation of Dot1l led to downregulation of direct MLL-AF9 targets and an MLL translocation-associated gene expression signature, whereas global gene expression remained largely unaffected. Suppression of MLL translocation-associated gene expression corresponded with dependence of MLL-AF9 leukemia on Dot1l in vivo. These data point to DOT1L as a potential therapeutic target in MLL-rearranged leukemia.
Copyright © 2011 Elsevier Inc. All rights reserved.
Publication types
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Research Support, N.I.H., Extramural
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Research Support, Non-U.S. Gov't
MeSH terms
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Animals
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Apoptosis
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Cell Cycle
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Cell Differentiation
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Cell Transformation, Neoplastic / metabolism
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Cell Transformation, Neoplastic / pathology
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Gene Rearrangement / genetics*
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Genetic Loci / genetics
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Hematopoiesis
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Histone-Lysine N-Methyltransferase
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Histones / metabolism*
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Homeodomain Proteins / metabolism
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Humans
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Lysine / metabolism*
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Methylation
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Methyltransferases / metabolism*
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Mice
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Myeloid Ecotropic Viral Integration Site 1 Protein
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Myeloid Progenitor Cells / metabolism
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Myeloid Progenitor Cells / pathology
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Myeloid-Lymphoid Leukemia Protein / metabolism*
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Neoplasm Proteins / metabolism
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Oncogene Proteins, Fusion / metabolism
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Protein Processing, Post-Translational
Substances
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Histones
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Homeodomain Proteins
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MLL-AF9 fusion protein, human
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Myeloid Ecotropic Viral Integration Site 1 Protein
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Neoplasm Proteins
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Oncogene Proteins, Fusion
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homeobox protein HOXA9
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Myeloid-Lymphoid Leukemia Protein
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DOT1L protein, human
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Methyltransferases
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Histone-Lysine N-Methyltransferase
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Lysine
Associated data
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GEO/GSE25911
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GEO/GSE29130