RuvBl2 cooperates with Ets2 to transcriptionally regulate hTERT in colon cancer

FEBS Lett. 2011 Aug 4;585(15):2537-44. doi: 10.1016/j.febslet.2011.07.005. Epub 2011 Jul 13.

Abstract

Human cancers utilise telomerase to maintain telomeres and prohibit cell senescence. Human telomerase reverse transcriptase (hTERT), an essential component of this complex, is regulated at the level of gene transcription. Using SILAC-proteomic analysis and molecular studies, we identified the AAA+ ATPase, RuvBl2 as a transcriptional regulator of hTERT and established that this regulation is through cooperation with Ets-2. In colon cancer patients, nuclear expression of RuvBl2 associated with nuclear expression of hTERT, pEts2 and advanced nodal disease (P<0.01, P=0.05 and P=0.03 respectively, n=170). These data firmly implicate RuvBl2 in Ets2 mediated regulation of hTERT in colon cancer which has functional and clinical consequences.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • ATPases Associated with Diverse Cellular Activities
  • Carrier Proteins / physiology*
  • Colonic Neoplasms / chemistry
  • Colonic Neoplasms / genetics*
  • Colonic Neoplasms / pathology*
  • DNA Helicases / physiology*
  • Disease Progression
  • Gene Expression Regulation, Neoplastic*
  • Humans
  • Proteomics
  • Proto-Oncogene Protein c-ets-2 / physiology*
  • Telomerase / genetics*
  • Transcription, Genetic

Substances

  • Carrier Proteins
  • ETS2 protein, human
  • Proto-Oncogene Protein c-ets-2
  • TERT protein, human
  • Telomerase
  • ATPases Associated with Diverse Cellular Activities
  • DNA Helicases
  • RUVBL2 protein, human