EX-HOM (EXome HOMozygosity): a proof of principle

Hum Hered. 2011;72(1):45-53. doi: 10.1159/000330164. Epub 2011 Aug 18.

Abstract

Objective: We provide the proof of principle that exome sequencing of only two affected siblings born to first-cousin parents is capable of directly identifying a single candidate gene for an autosomal recessive disorder. This strategy, which we call EX-HOM (EXome HOMozygosity), combines in a single step the capacity of exome sequencing to identify all the coding variants present in a genome with the property of homozygosity mapping to limit the search for candidate genes to specific chromosomal regions.

Methods: We sequenced the exomes of two siblings born to first-cousin parents affected with dysmyelinating leukodystrophy and spastic paraparesis caused by a mutation in FA2H. We used exome sequencing data to identify homozygous regions shared by the two affected siblings (EX-HOM regions), compared them with the regions of maximum LOD score obtained with SNP genotyping, and selected the candidate variants within.

Results: We identified regions of shared homozygosity (>1 Mb) accounting for about 290 Mb, containing only 3 candidate variants. Among these, the FA2H mutation remained the only plausible one.

Conclusion: In single consanguineous pedigrees with a few affected sibs, EX-HOM can be a one-step approach to identify the candidate genetic defect, bypassing obstacles such as genetic heterogeneity and the need for large pedigrees.

Publication types

  • Evaluation Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Exons / genetics*
  • Genes, Recessive / genetics*
  • Genetic Association Studies / methods*
  • Genetic Variation*
  • Hereditary Central Nervous System Demyelinating Diseases / genetics*
  • Homozygote*
  • Humans
  • Italy
  • Lod Score
  • Mixed Function Oxygenases / genetics
  • Paraparesis, Spastic / genetics*
  • Pedigree
  • Polymorphism, Single Nucleotide / genetics
  • Sequence Analysis, DNA
  • Siblings

Substances

  • Mixed Function Oxygenases
  • fatty acid alpha-hydroxylase