Neural cell adhesion molecule differentially interacts with isoforms of the fibroblast growth factor receptor

Neuroreport. 2011 Oct 26;22(15):727-32. doi: 10.1097/WNR.0b013e3283491682.

Abstract

The fibroblast growth factor receptor (FGFR) can be activated through direct interactions with various fibroblast growth factors or through a number of cell adhesion molecules, including the neural cell adhesion molecule (NCAM). We produced recombinant proteins comprising the ligand-binding immunoglobulin-like modules 2 and 3 of FGFR1b, FGFR1c, FGFR2b, FGFR2c, FGFR3b, FGFR3c, and FGFR4, and found that all FGFR isoforms, except for FGFR4, interacted with NCAM. The binding affinity of NCAM-FGFR interactions was considerably higher for splice variant 'b' than for splice variant 'c'. We suggest that the expression pattern of various FGFR isoforms determines the cell context-specific effects of NCAM signaling through FGFR.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Male
  • Neural Cell Adhesion Molecules / metabolism*
  • Protein Isoforms / metabolism
  • Rats
  • Rats, Wistar
  • Receptors, Fibroblast Growth Factor / metabolism*
  • Recombinant Proteins / metabolism
  • Signal Transduction / physiology*
  • Surface Plasmon Resonance

Substances

  • Neural Cell Adhesion Molecules
  • Protein Isoforms
  • Receptors, Fibroblast Growth Factor
  • Recombinant Proteins