Assessment of ¹⁸⁸Re marked anti MHC class II antibody by peripheral blood mononuclear cells stimulated by donor alloantigen

Chin Med J (Engl). 2011 Aug;124(16):2512-6.

Abstract

Background: Previous studies showed that anti MHC-II monoclone antibody (MAb) only had partial inhibiting effect of alloreactive mixed lymphocyte reaction (MLR) in vitro and it was unsteady and non-persistent. The aim of this research was to determine whether radioactive isotope (188)Re marked MHC-II antibody could benefit the allograft acceptance in transplantation as compared to normal MHC-II antibody.

Methods: 188Re was incorporated to 2E9/13F (ab')(2) which is against swine MHC class II antigen (MAb-(188)Re). Porcine peripheral blood mononuclear (PBMC) cells were examined for proliferation and cytokine mRNA expression after stimulation with MHC-II MAb or MAb-(188)Re.

Results: The proliferative response of recipient PBMCs in mixed lymphocyte reaction (MLR) to donor alloantigen showed that the stimulation index of MAb-(188)Re group was significantly lower than the MHC-II MAb group and control (P < 0.05). mRNA expression of interleukin 2, interferon Υ and tumor necrosis factor α (type 1 cytokines) was lower in MAb-(188)Re group than the MHC-II MAb group, while interleukin 10 (type 2 cytokines) was higher in MAb-(188)Re group in the first 24 hours.

Conclusion: MAb-(188)Re could help the graft acceptance by inhibiting T cell proliferation, lowering the expression of type 1 cytokines and elevating the type 2 cytokines produced by PBMC.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Antibodies, Monoclonal / chemistry
  • Antibodies, Monoclonal / pharmacology*
  • Cell Proliferation / drug effects
  • Interleukin-10 / genetics
  • Interleukin-2 / genetics
  • Isoantigens / immunology*
  • Leukocytes, Mononuclear / drug effects*
  • Leukocytes, Mononuclear / radiation effects
  • Lymphocyte Culture Test, Mixed
  • Mitomycin / pharmacology
  • Radioisotopes*
  • Reverse Transcriptase Polymerase Chain Reaction
  • Rhenium*
  • Swine
  • Tumor Necrosis Factor-alpha / genetics

Substances

  • Antibodies, Monoclonal
  • Interleukin-2
  • Isoantigens
  • Radioisotopes
  • Tumor Necrosis Factor-alpha
  • Interleukin-10
  • Mitomycin
  • Rhenium