Transcriptional control of c-myc gene expression during stimulation of murine B lymphocytes

J Immunol. 1990 Jul 15;145(2):732-6.

Abstract

The binding of ligand to surface IgR results in the initial activation of B cells. As shown by experiments in which B cells are polyclonally stimulated with anti-Ig antibody, this includes an early increase in c-myc gene expression. In our study a correlation between increases in the rate of gene transcription and the level of c-myc mRNA was observed both with the brief increase in c-myc expression that is induced by anti-Ig, as well as with the more intense and prolonged expression of c-myc that follows treatment with anti-Ig plus the cytoskeleton perturbing agent, cytochalasin. Elevation of the rate of initiation was detected with both stimulatory regimens although the combination of anti-Ig plus cytochalasin increased the rate of elongation in addition to the rate of initiation. These results suggest that stimulation of B lymphocytes alters expression of trans-acting factors that regulate transcription of the c-myc gene.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • B-Lymphocytes / physiology*
  • Cytochalasin D / pharmacology
  • Gene Expression Regulation / drug effects
  • In Vitro Techniques
  • Mice
  • Proto-Oncogene Proteins / genetics*
  • Proto-Oncogene Proteins c-myc
  • RNA, Messenger / genetics
  • Receptors, Antigen, B-Cell / physiology
  • Restriction Mapping
  • Time Factors
  • Transcription, Genetic / drug effects

Substances

  • Proto-Oncogene Proteins
  • Proto-Oncogene Proteins c-myc
  • RNA, Messenger
  • Receptors, Antigen, B-Cell
  • Cytochalasin D