Millimeter wave radiation induces apoptosis via affecting the ratio of Bax/Bcl-2 in SW1353 human chondrosarcoma cells

Oncol Rep. 2012 Mar;27(3):664-72. doi: 10.3892/or.2011.1558. Epub 2011 Nov 22.

Abstract

The efficacy and safety of millimeter wave radiation has been proven for various types of malignant tumors. However, the mechanisms underlying effects of millimeter wave radiation on apoptosis are still unclear. The present study was undertaken to examine the effects of millimeter wave radiation on cell apoptosis and mitochondrial membrane potential, and to determine the molecular mechanism of millimeter wave radiation-induced apoptosis by investigating the expression of Bcl-2 family proteins (Bcl-2, Bax), caspase-9 and caspase-3 in SW1353 cells. We found that millimeter wave radiation suppressed the viability of SW1353 cells, demonstrating that millimeter wave radiation induced cell apoptosis and reduced cell viability in a time-dependent manner. Furthermore, we observed that treatment of cells with millimeter wave radiation significantly induced loss of mitochondrial membrane potential, upregulated proapoptotic Bax, caspase-9 and caspase-3, but did not significantly change levels of antiapoptotic Bcl-2. These data suggested that millimeter wave radiation may induce apoptosis via affecting the ratio of Bax/Bcl-2 in SW1353 cells.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Apoptosis / genetics
  • Apoptosis / radiation effects*
  • Bone Neoplasms / genetics
  • Bone Neoplasms / metabolism
  • Bone Neoplasms / pathology
  • Bone Neoplasms / radiotherapy*
  • Caspase 3 / metabolism
  • Caspase 9 / metabolism
  • Caspase Inhibitors
  • Cell Survival / radiation effects
  • Chondrosarcoma / genetics
  • Chondrosarcoma / metabolism
  • Chondrosarcoma / pathology
  • Chondrosarcoma / radiotherapy*
  • Enzyme Activation / radiation effects
  • Gene Expression / radiation effects
  • Humans
  • Membrane Potential, Mitochondrial / radiation effects
  • Proto-Oncogene Proteins c-bcl-2 / genetics
  • Proto-Oncogene Proteins c-bcl-2 / metabolism*
  • Tumor Cells, Cultured
  • Up-Regulation / radiation effects

Substances

  • Caspase Inhibitors
  • Proto-Oncogene Proteins c-bcl-2
  • Caspase 3
  • Caspase 9