Cyclosporine immunomodulation retards regeneration of surgically transected corneal nerves

Invest Ophthalmol Vis Sci. 2012 Feb 13;53(2):732-40. doi: 10.1167/iovs.11-8445. Print 2012 Feb.

Abstract

Purpose: To determine whether immunomodulation with cyclosporine (CsA) affects reinnervation after surgical transection of stromal nerves.

Methods: Thy1-YFP+ neurofluorescent mice underwent lamellar corneal surgery and 3 days later, received artificial tears or CsA eye drops for 6 weeks. Serial in vivo wide-field stereofluorescent microscopy was performed to determine changes in nerve fiber density (NFD). Real-time quantitative PCR was performed to determine the expression of neurotrophins and cytokines (IL6 and TNF-α). Compartmental culture of trigeminal ganglion neurons was performed in Campenot devices to determine whether CsA directly affects neurite outgrowth.

Results: Yellow fluorescent protein (YFP)-positive cells significantly increased at 3 and 7 days after surgery. The number of YFP-positive cells in the cornea was significantly lower in the CsA group than that in the control group. The percentage increase in NFD between 2 to 6 weeks was greater in the control group (80% ± 10%, P = 0.05) than that in the CsA group (39% ± 21%). The CsA group also exhibited lower expression of IL6 and TNF-α (P = 0.01). In compartmental culture experiments, neurite outgrowth toward side compartments containing CsA was significantly less (2.29 ± 0.4 mm, P = 0.01) than that toward side compartments containing vehicle (3.97 ± 0.71 mm).

Conclusions: Immunomodulation with CsA reduces the expression of cytokines (IL6) in the cornea and retards regenerative sprouting from transected corneal stromal nerve trunks. In addition, CsA has a direct growth inhibitory action on neurites as well.

Publication types

  • Randomized Controlled Trial
  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Cornea / innervation*
  • Cornea / physiology*
  • Cornea / surgery
  • Corneal Stroma / innervation
  • Cyclosporine / pharmacology*
  • Immunosuppressive Agents / pharmacology*
  • Interleukin-6 / metabolism
  • Mice
  • Nerve Growth Factors / metabolism
  • Neurites / metabolism
  • Real-Time Polymerase Chain Reaction
  • Regeneration / drug effects*
  • Tumor Necrosis Factor-alpha / metabolism

Substances

  • Immunosuppressive Agents
  • Interleukin-6
  • Nerve Growth Factors
  • Tumor Necrosis Factor-alpha
  • Cyclosporine