Phenotypic and functional maturation of murine dendritic cells (DCs) induced by purified Glycyrrhizin (GL)

Int Immunopharmacol. 2012 Mar;12(3):518-25. doi: 10.1016/j.intimp.2012.01.006. Epub 2012 Jan 29.

Abstract

The aim of this study is to investigate phenotypic and functional modulation of murine dendritic cells (DCs) with use of purified Glycyrrhizin (GL). These impacts of GL on DCs both from bone marrow derived DCs and established DC cell 2.4 were assessed with conventional scanning electron microscopy (SEM), flow cytometry (FCM), transmission electron microscopy (TEM), cytochemistry assay, FITC-dextran, bio-assay and enzyme linked immunosorbent assay (ELISA). We found that the purified GL induced phenotypic maturation as evidenced by increased expression of CD86, CD40, CD80, CD83 and major histocompatibility complex II (MHC II). The functional tests showed the activity of acidic phosphatase (ACP) inside the DCs2.4 cells were down- regulated after treatment with GL (which occurs when phagocytosis of DCs2.4 cells were decreased). Finally, we proved that GL increased the production of IL-12, IL-10 and decreased the production of tumor necrosis factor alpha (TNF-α). These data indicated that GL could promote maturation of DCs and this adjuvant-like activity may have potential therapeutic value. It is therefore concluded that GL could exert positive modulation on murine DCs.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Acid Phosphatase / metabolism
  • Animals
  • Bone Marrow Cells
  • Cell Line
  • Cell Proliferation / drug effects
  • Cytoplasmic Vesicles / drug effects
  • Cytoplasmic Vesicles / ultrastructure
  • Dendritic Cells / drug effects*
  • Dendritic Cells / ultrastructure
  • Enzyme Activation / physiology
  • Enzyme-Linked Immunosorbent Assay
  • Female
  • Flow Cytometry
  • Glycyrrhizic Acid / pharmacology*
  • Interleukin-10 / biosynthesis
  • Interleukin-12 / biosynthesis
  • Mice
  • Mice, Inbred C57BL
  • Microscopy, Electron, Scanning
  • Phagocytosis / drug effects
  • Phenotype
  • Pinocytosis / drug effects
  • Receptors, Tumor Necrosis Factor / biosynthesis
  • Receptors, Tumor Necrosis Factor / genetics
  • Tumor Necrosis Factor-alpha / biosynthesis

Substances

  • Receptors, Tumor Necrosis Factor
  • Tumor Necrosis Factor-alpha
  • Interleukin-10
  • Interleukin-12
  • Glycyrrhizic Acid
  • Acid Phosphatase