FDG-PET as a pharmacodynamic biomarker for early assessment of treatment response to linifanib (ABT-869) in a non-small cell lung cancer xenograft model

Cancer Chemother Pharmacol. 2012 Jun;69(6):1669-72. doi: 10.1007/s00280-012-1840-z. Epub 2012 Feb 12.

Abstract

Linifanib (ABT-869) is a multitargeted receptor tyrosine kinase inhibitor. This work aims to evaluate F-fluorodeoxyglucose-positron emission tomography (FDG-PET) as a pharmacodynamic (PD) biomarker for linifanib treatment utilizing the Calu-6 model of human non-small cell lung (NSCLC) cancer in SCID-beige mice. Animals received either vehicle or 12.5 mg/kg linifanib orally twice a day for the duration of the study. Imaging was performed at -1, 1, 3, and 7 days after beginning treatment (n = 12-14 per group). Linifanib inhibited tumor growth and suppressed tumor metabolic activity. Changes in tumor FDG uptake were observed as early as 1 day after beginning linifanib treatment and were sustained for the duration of the study. This study confirms that linifanib is efficacious in this xenograft model of human NSCLC and confirms FDG-PET is a potential PD biomarker strategy for linifanib therapy.

MeSH terms

  • Animals
  • Carcinoma, Non-Small-Cell Lung / diagnostic imaging
  • Carcinoma, Non-Small-Cell Lung / drug therapy*
  • Cell Line, Tumor
  • Fluorodeoxyglucose F18*
  • Humans
  • Indazoles / therapeutic use*
  • Lung Neoplasms / diagnostic imaging
  • Lung Neoplasms / drug therapy*
  • Mice
  • Mice, SCID
  • Phenylurea Compounds / therapeutic use*
  • Positron-Emission Tomography*
  • Receptor Protein-Tyrosine Kinases / antagonists & inhibitors*
  • Xenograft Model Antitumor Assays

Substances

  • Indazoles
  • Phenylurea Compounds
  • Fluorodeoxyglucose F18
  • linifanib
  • Receptor Protein-Tyrosine Kinases