CTCF binds to sites in the major histocompatibility complex that are rapidly reconfigured in response to interferon-gamma

Nucleic Acids Res. 2012 Jul;40(12):5262-70. doi: 10.1093/nar/gks158. Epub 2012 Feb 25.

Abstract

Activation of the major histocompatibility complex (MHC) by interferon-gamma (IFN-γ) is a fundamental step in the adaptive immune response to pathogens. Here, we show that reorganization of chromatin loop domains in the MHC is evident within the first 30 min of IFN-γ treatment of fibroblasts, and that further dynamic alterations occur up to 6 h. These very rapid changes occur at genomic sites which are occupied by CTCF and are close to IFN-γ-inducible MHC genes. Early responses to IFN-γ are thus initiated independently of CIITA, the master regulator of MHC class II genes and prepare the MHC for subsequent induction of transcription.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Binding Sites
  • CCCTC-Binding Factor
  • Cells, Cultured
  • Chromatin / chemistry
  • Chromatin / drug effects
  • Humans
  • Interferon-gamma / pharmacology*
  • Major Histocompatibility Complex*
  • Matrix Attachment Regions / drug effects
  • Repressor Proteins / metabolism*
  • Transcription Factors / metabolism

Substances

  • CCCTC-Binding Factor
  • CTCF protein, human
  • Chromatin
  • Repressor Proteins
  • Transcription Factors
  • Interferon-gamma