Norepinephrine suppresses IFN-γ and TNF-α production by murine intestinal intraepithelial lymphocytes via the β₁ adrenoceptor

J Neuroimmunol. 2012 Apr;245(1-2):66-74. doi: 10.1016/j.jneuroim.2012.02.007. Epub 2012 Mar 6.

Abstract

We examined whether norepinephrine (NE) had direct effects on cytokine production by murine intestinal intraepithelial lymphocytes (IELs), compared with splenocytes. CD3⁺ IELs and CD3⁺ splenocytes expressed α(1B), α(1D), α(2C), β₁, β₂, and β₃ adrenoceptors (ARs). NE significantly suppressed IFN-γ and TNF-α production by IELs and splenocytes ex vivo. The suppressive effects of NE in IELs were reversed by β₁ AR antagonist CGP-20712A, whereas those in splenocytes were reversed by β₂ AR antagonist ICI118,551. In IELs, β₁ AR agonist xamoterol mimicked the suppressive effects of NE. These results indicated NE regulates intestinal mucosal immune responses mediated by IELs via β₁ AR.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Immunity, Mucosal / immunology*
  • Interferon-gamma / antagonists & inhibitors*
  • Interferon-gamma / biosynthesis
  • Intestinal Mucosa / cytology
  • Intestinal Mucosa / immunology*
  • Intestinal Mucosa / metabolism
  • Lymphocyte Subsets / immunology*
  • Lymphocyte Subsets / metabolism
  • Male
  • Mice
  • Mice, Inbred C57BL
  • Primary Cell Culture
  • Receptors, Adrenergic, beta-1 / physiology*
  • Spleen / cytology
  • Spleen / immunology
  • Tumor Necrosis Factor-alpha / antagonists & inhibitors*
  • Tumor Necrosis Factor-alpha / biosynthesis

Substances

  • Adrb1 protein, mouse
  • Receptors, Adrenergic, beta-1
  • Tumor Necrosis Factor-alpha
  • Interferon-gamma