Inhibition of miR-29 by TGF-beta-Smad3 signaling through dual mechanisms promotes transdifferentiation of mouse myoblasts into myofibroblasts

PLoS One. 2012;7(3):e33766. doi: 10.1371/journal.pone.0033766. Epub 2012 Mar 16.

Abstract

MicroRNAs (miRNAs) are non-coding RNAs that regulate gene expression in post-transcriptional fashion, and emerging studies support their importance in regulating many biological processes, including myogenic differentiation and muscle development. miR-29 is a promoting factor during myogenesis but its full spectrum of impact on muscle cells has yet to be explored. Here we describe an analysis of miR-29 affected transcriptome in C2C12 muscle cells using a high throughput RNA-sequencing platform. The results reveal that miR-29 not only functions to promote myogenic differentiation but also suppresses the transdifferentiation of myoblasts into myofibroblasts. miR-29 inhibits the fibrogenic differentiation through down-regulating both extracellular matrix genes and cell adhesion genes. We further demonstrate that miR-29 is under negative regulation by TGF-beta (TGF-β)-Smad3 signaling via dual mechanisms of both inhibiting MyoD binding and enhancing Yin Yang 1 (YY1)-recruited Polycomb association. Together, these results identify miR-29 as a pleiotropic molecule in both myogenic and fibrogenic differentiation of muscle cells.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Base Sequence
  • Cell Line
  • Cell Transdifferentiation / genetics
  • Cell Transdifferentiation / physiology
  • Down-Regulation
  • Mice
  • MicroRNAs / antagonists & inhibitors*
  • MicroRNAs / genetics*
  • MicroRNAs / metabolism
  • Models, Biological
  • MyoD Protein / metabolism
  • Myoblasts, Skeletal / cytology*
  • Myoblasts, Skeletal / metabolism*
  • Myofibroblasts / cytology*
  • Myofibroblasts / metabolism*
  • Polycomb-Group Proteins
  • Promoter Regions, Genetic
  • RNA, Small Interfering / genetics
  • Repressor Proteins / metabolism
  • Signal Transduction / physiology
  • Smad3 Protein / antagonists & inhibitors
  • Smad3 Protein / genetics
  • Smad3 Protein / metabolism*
  • Transcriptome
  • Transforming Growth Factor beta / metabolism*
  • YY1 Transcription Factor / metabolism

Substances

  • MIRN29 microRNA, mouse
  • MicroRNAs
  • MyoD Protein
  • MyoD1 myogenic differentiation protein
  • Polycomb-Group Proteins
  • RNA, Small Interfering
  • Repressor Proteins
  • Smad3 Protein
  • Smad3 protein, mouse
  • Transforming Growth Factor beta
  • YY1 Transcription Factor
  • Yy1 protein, mouse