Early impairment of long-term depression in the perirhinal cortex of a mouse model of Alzheimer's disease

Rejuvenation Res. 2012 Apr;15(2):231-4. doi: 10.1089/rej.2011.1311.

Abstract

Visual recognition memory is early impaired in Alzheimer's disease. Long-term depression of synaptic transmission in the perirhinal cortex is critically involved in this form of memory. We found that synaptic transmission was impaired in perirhinal cortex slices obtained from 3-month-old Tg2576 mice, and that 3,000 pulses at 5 Hz induced long-term depression in perirhinal cortex slices from age-matched control mice, but not in those from Tg2576 mice. To our knowledge, these data provide the first evidence of synaptic transmission and long-term depression impairment in the perirhinal cortex in an animal model of Alzheimer's disease, and the earliest synaptic deficit in Tg2576 mice.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Alzheimer Disease / genetics*
  • Alzheimer Disease / physiopathology*
  • Amyloid beta-Peptides / genetics
  • Animals
  • Brain Mapping / methods
  • Cerebral Cortex / physiopathology*
  • Disease Models, Animal
  • Electrophysiology / methods
  • Long-Term Synaptic Depression*
  • Memory
  • Mice
  • Mice, Transgenic
  • Neuronal Plasticity*
  • Synaptic Transmission
  • Time Factors

Substances

  • Amyloid beta-Peptides