Abstract
OPCML, frequently inactivated in ovarian tumors, mediates its antitumor effect via binding to the extracellular domains of several important oncogenic receptor tyrosine kinases (RTK). This, in turn, leads to the downregulation of RTKs in tumor cells and results in significant inhibition of tumor growth.
©2012 AACR.
MeSH terms
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Animals
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Carcinoma, Ovarian Epithelial
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Cell Adhesion Molecules / metabolism*
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Female
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GPI-Linked Proteins / metabolism
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Humans
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Neoplasms, Glandular and Epithelial / metabolism*
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Ovarian Neoplasms / metabolism*
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Receptor Protein-Tyrosine Kinases / metabolism*
Substances
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Cell Adhesion Molecules
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GPI-Linked Proteins
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OPCML protein, human
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Receptor Protein-Tyrosine Kinases