NMDA depolarizations and long-term potentiation are reduced in the aged rat neocortex

Brain Res. 1990 Oct 15;530(1):142-6. doi: 10.1016/0006-8993(90)90671-w.

Abstract

N-Methyl-D-aspartate (NMDA) responses were recorded intracellularly in layer V neocortical neurons in in vitro slices taken from young (4-6 months) and aged (27-29 months) Fischer 344 rats. Increasing amounts of NMDA produced membrane depolarizations in both groups of cells. The regression analysis showed significantly reduced sensitivity to NMDA in old neurons compared to young. A significant long-term potentiation of the field potential evoked by subcortical white matter stimulation was present in young but not in old slices. These results suggest that aging results in a decreased sensitivity to NMDA and impaired synaptic plasticity in the neocortex.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Aging / physiology*
  • Alzheimer Disease / physiopathology*
  • Animals
  • Cerebral Cortex / drug effects*
  • Disease Models, Animal
  • Electric Stimulation
  • In Vitro Techniques
  • Membrane Potentials / drug effects
  • N-Methylaspartate / pharmacology*
  • Neurons / drug effects*
  • Rats
  • Rats, Inbred F344
  • Time Factors

Substances

  • N-Methylaspartate