Pyridostatin analogues promote telomere dysfunction and long-term growth inhibition in human cancer cells

Org Biomol Chem. 2012 Aug 28;10(32):6537-46. doi: 10.1039/c2ob25830g. Epub 2012 Jul 13.

Abstract

The synthesis, biophysical and biological evaluation of a series of G-quadruplex interacting small molecules based on a N,N'-bis(quinolinyl)pyridine-2,6-dicarboxamide scaffold is described. The synthetic analogues were evaluated for their ability to stabilize telomeric G-quadruplex DNA, some of which showed very high stabilization potential associated with high selectivity over double-stranded DNA. The compounds exhibited growth arrest of cancer cells with detectable selectivity over normal cells. Long-time growth arrest was accompanied by senescence, where telomeric dysfunction is a predominant mechanism together with the accumulation of restricted DNA damage sites in the genome. Our data emphasize the potential of a senescence-mediated anticancer therapy through the use of G-quadruplex targeting small molecules based on the molecular framework of pyridostatin.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Aminoquinolines / chemistry
  • Aminoquinolines / pharmacology*
  • Antineoplastic Agents / chemistry
  • Antineoplastic Agents / pharmacology*
  • Base Sequence
  • Cell Line, Tumor
  • Cell Survival / drug effects
  • G-Quadruplexes / drug effects
  • Humans
  • Models, Biological
  • Molecular Sequence Data
  • Molecular Structure
  • Picolinic Acids / chemistry
  • Picolinic Acids / pharmacology*
  • Static Electricity
  • Telomere / drug effects*

Substances

  • Aminoquinolines
  • Antineoplastic Agents
  • Picolinic Acids
  • pyridostatin