Changes in the immune and endocrine responses of patients with pulmonary tuberculosis undergoing specific treatment

Ann N Y Acad Sci. 2012 Jul:1262:10-5. doi: 10.1111/j.1749-6632.2012.06643.x.

Abstract

We evaluated immune and endocrine status following antituberculosis treatment in HIV-negative patients with newly diagnosed tuberculosis (TB). Treatment led to a decrease in IL-6, IL-1β, and C-reactive protein levels. Cortisol levels decreased throughout the anti-TB treatment, particularly after 4 months, but changes were less pronounced than those seen in proinflammatory mediators. Specific therapy resulted in increased dehydroepiandrosterone (DHEA) levels, which peaked after 4 months and started to decline after 6 months of treatment, reaching levels below those detected at inclusion. In contrast, in most patients, dehydroepiandrosterone sulfate (DHEAS) levels remained unchanged, although a trend toward increased concentrations was observed in a few cases 3 months after the treatment was finished. Specific therapy also resulted in more balanced cortisol/DHEA and cortisol/DHEAS ratios. Etiologic treatment involves favorable immune and endocrine changes, which may account for its beneficial effects.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adrenal Cortex Hormones / blood*
  • Adult
  • Antitubercular Agents / therapeutic use
  • C-Reactive Protein / metabolism
  • Case-Control Studies
  • Dehydroepiandrosterone / blood
  • Dehydroepiandrosterone Sulfate / blood
  • Female
  • Humans
  • Hydrocortisone / blood
  • Inflammation Mediators / blood*
  • Interleukin-1beta / blood
  • Interleukin-6 / blood
  • Male
  • Middle Aged
  • Tuberculosis, Pulmonary / blood*
  • Tuberculosis, Pulmonary / drug therapy
  • Tuberculosis, Pulmonary / immunology*
  • Young Adult

Substances

  • Adrenal Cortex Hormones
  • Antitubercular Agents
  • IL6 protein, human
  • Inflammation Mediators
  • Interleukin-1beta
  • Interleukin-6
  • Dehydroepiandrosterone
  • Dehydroepiandrosterone Sulfate
  • C-Reactive Protein
  • Hydrocortisone