Factor VIIa-antithrombin complexes in patients with non-neoplastic portal vein thrombosis with and without cirrhosis

Int J Lab Hematol. 2013 Feb;35(1):101-5. doi: 10.1111/ijlh.12003. Epub 2012 Sep 10.

Abstract

Introduction: Portal vein thrombosis (PVT) is caused by local and systemic prothrombotic risk factors. In this case-control study, we evaluated the use of the Factor VIIa-antithrombin complex (FVIIa-AT) complex assay as a hypercoagulability marker in patients with PVT.

Methods: Two different groups of cases were considered: (i) n = 12 noncirrhotic PVT patients, (ii) n = 33 cirrhotic patients with PVT. Controls were sex and age-matched healthy volunteers and cirrhotic subjects without PVT, respectively.

Results: Levels of the FVIIa-AT complex were significantly higher in noncirrhotic PVT subjects (132 ± 32 pM) than in healthy volunteers (108 ± 18 pM, P = 0.04). No significant difference in FVIIa-AT complexes was seen between cirrhotic patients with (64 ± 20 pM) or without (61 ± 24 pM) PVT. A linear correlation was seen between FVIIa-AT and FVIIa in noncirrhotic PVT subjects. In cirrhotic patients, FVIIa-AT complexes depended on both FVIIa and AT.

Conclusion: These results confirm the utility of the FVIIa-AT assay in identifying the hypercoagulable state of noncirrhotic patients because of a previous thrombotic event.

MeSH terms

  • Adult
  • Aged
  • Antithrombin III / analysis*
  • Biomarkers / blood
  • Blood Coagulation
  • Case-Control Studies
  • Enzyme-Linked Immunosorbent Assay
  • Factor VIIa / analysis*
  • Female
  • Humans
  • Liver Cirrhosis / complications*
  • Male
  • Middle Aged
  • Portal Vein / pathology*
  • Retrospective Studies
  • Thrombophilia / physiopathology
  • Up-Regulation*
  • Venous Thrombosis / blood*
  • Venous Thrombosis / complications
  • Venous Thrombosis / pathology
  • Venous Thrombosis / physiopathology
  • Young Adult

Substances

  • Biomarkers
  • Antithrombin III
  • Factor VIIa