Avidity of influenza-specific memory CD8+ T-cell populations decays over time compromising antiviral immunity

Eur J Immunol. 2012 Dec;42(12):3235-42. doi: 10.1002/eji.201242575. Epub 2012 Oct 16.

Abstract

Decline of cell-mediated immunity is often attributed to decaying T-cell numbers and their distribution in peripheral organs. This study examined the hypothesis that qualitative as well as quantitative changes contribute to the declining efficacy of CD8(+) T-cell memory. Using a model of influenza virus infection, where loss of protective CD8(+) T-cell immunity was observed 6 months postinfection, we found no decline in antigen-specific T-cell numbers or migration to the site of secondary infection. There was, however, a large reduction in antigen-specific CD8(+) T-cell degranulation, cytokine secretion, and polyfunctionality. A profound loss of high-avidity T cells over time indicated that failure to confer protective immunity resulted from the inferior functional capacity of remaining low avidity cells. These data imply that high-avidity central memory T cells wane with declining antigen levels, leaving lower avidity T cells with reduced functional capabilities.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Alphainfluenzavirus / immunology*
  • Alphainfluenzavirus / metabolism
  • Animals
  • CD8-Positive T-Lymphocytes
  • Cell Degranulation / immunology*
  • Cell Movement / immunology*
  • Cytokines / immunology
  • Cytokines / metabolism
  • Immunity, Cellular*
  • Immunologic Memory*
  • Mice
  • Orthomyxoviridae Infections / immunology*
  • Orthomyxoviridae Infections / metabolism
  • Time Factors

Substances

  • Cytokines