MiR-136 promotes apoptosis of glioma cells by targeting AEG-1 and Bcl-2

FEBS Lett. 2012 Oct 19;586(20):3608-12. doi: 10.1016/j.febslet.2012.08.003. Epub 2012 Aug 9.

Abstract

MicroRNAs have the capacity to coordinately repress multiple target genes and interfere with biological functions of the cell, such as proliferation and apoptosis. Here we report that miR-136 is downregulated in human glioma, and that the miRNA promotes apoptosis of glioma cells induced by chemotherapy. Two anti-apoptotic genes, AEG-1 and Bcl-2, are identified as targets of miR-136, and restoration of AEG-1 or Bcl-2 expression suppresses miR-136-enhanced apoptosis. Therefore, miR-136 might play a tumor-suppressive role in human glioma and thereby might represent a potential therapeutic strategy.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Apoptosis / drug effects
  • Apoptosis / genetics*
  • Base Sequence
  • Cell Adhesion Molecules / genetics*
  • Cisplatin / pharmacology
  • Down-Regulation / drug effects
  • Down-Regulation / genetics
  • Glioma / genetics*
  • Glioma / pathology*
  • Humans
  • Membrane Proteins
  • MicroRNAs / genetics*
  • MicroRNAs / metabolism*
  • Proto-Oncogene Proteins c-bcl-2 / genetics*
  • RNA-Binding Proteins

Substances

  • Cell Adhesion Molecules
  • MIRN136 microRNA, human
  • MTDH protein, human
  • Membrane Proteins
  • MicroRNAs
  • Proto-Oncogene Proteins c-bcl-2
  • RNA-Binding Proteins
  • Cisplatin