[Simultaneous inhibition of XIAP and survivin expression on EMT and invasion of human pancreatic cancer cells]

Zhong Nan Da Xue Xue Bao Yi Xue Ban. 2012 Sep;37(9):883-8. doi: 10.3969/j.issn.1672-7347.2012.09.004.
[Article in Chinese]

Abstract

Objective: To investigate the simultaneous inhibition of X-linked inhibitor of apoptosis protein (XIAP) and survivin expression on epithelial-mesenchymal transition (EMT) and invasiion of pancreatic cancer cells Panc-1, and its mechanism.

Methods: On the established human pancreatic cancer cells Panc-1-XS, the expression of XIAP and survivin was inhibited simultaneously. Cell invasion and migration were detected by Transwell chamber experiments and scratch test, and the expression of epithelial marker E-cadherin, mesenchymal markers Slug, phosphatase and tensin homolog deleted on chromosome ten (PTEN) and P-Akt protein was determined by Western blot.

Results: Cell invasion and migration of Panc-1-XS cells decreased significantly, accompanied by significantly upregulated protein expression of E-cadherin, and significantly declined protein expression of the Slug, indicating increased mesenchymal-epithelial conversion (MET); and increased protein expression of PTEN, and declined protein expression of P-Akt.

Conclusion: Simultaneously inhibiting the expression of XIAP and survivin can partially reverse EMT phenotype of pancreatic cancer Panc-1 cells, which then significantly reduces the cell invasion and migration of Panc-1 cell lines. This process may be regulated by PTEN/PI3K/Akt signaling pathway.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Antigens, CD
  • Cadherins / metabolism
  • Cell Line, Tumor
  • Cell Movement
  • Epithelial-Mesenchymal Transition / genetics*
  • Humans
  • Inhibitor of Apoptosis Proteins / genetics
  • Inhibitor of Apoptosis Proteins / metabolism*
  • Neoplasm Invasiveness
  • PTEN Phosphohydrolase / metabolism
  • Pancreatic Neoplasms / pathology*
  • Proto-Oncogene Proteins c-akt / metabolism
  • Signal Transduction
  • Snail Family Transcription Factors
  • Survivin
  • Transcription Factors / metabolism
  • X-Linked Inhibitor of Apoptosis Protein / genetics
  • X-Linked Inhibitor of Apoptosis Protein / metabolism*

Substances

  • Antigens, CD
  • BIRC5 protein, human
  • CDH1 protein, human
  • Cadherins
  • Inhibitor of Apoptosis Proteins
  • SNAI1 protein, human
  • Snail Family Transcription Factors
  • Survivin
  • Transcription Factors
  • X-Linked Inhibitor of Apoptosis Protein
  • XIAP protein, human
  • Proto-Oncogene Proteins c-akt
  • PTEN Phosphohydrolase