Inhibition by anti-HLA class I mAb of IL-2 and IL-2 receptor synthesis in lymphocytes stimulated with PHA-P

Cell Immunol. 1990 Apr 1;126(2):420-7. doi: 10.1016/0008-8749(90)90333-m.

Abstract

Inhibition by anti-HLA Class I monoclonal antibody (mAb) Q6/64 of phytohemagglutinin (PHA)-P-induced peripheral blood mononuclear cell (PMBC) proliferation is associated with a reduction of Tac expression and interleukin 2 (IL-2) secretion. To analyze the mechanism(s) underlying the latter phenomena, the Tac gene and IL-2 gene transcription was analyzed by a nuclear transcription assay. No synthesis of Tac and IL-2 mRNA was detected in PBMC stimulated with PHA-P in the presence of mAb Q6/64. In conjunction with our recently published data, these results indicate that the blocking by anti-HLA Class I mAb of PHA-P-induced PBMC proliferation reflects an inhibitory effect within the signal transduction pathway leading to transcriptional activation of IL-2 and IL-2 receptor genes.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Antibodies, Monoclonal / immunology*
  • Histocompatibility Antigens Class I / immunology*
  • Humans
  • Interleukin-2 / biosynthesis*
  • Interleukin-2 / genetics
  • Lymphocyte Activation
  • Lymphocytes / immunology*
  • Phytohemagglutinins / pharmacology*
  • RNA, Messenger / analysis
  • Receptors, Interleukin-2 / biosynthesis*
  • Receptors, Interleukin-2 / genetics

Substances

  • Antibodies, Monoclonal
  • Histocompatibility Antigens Class I
  • Interleukin-2
  • Phytohemagglutinins
  • RNA, Messenger
  • Receptors, Interleukin-2
  • phytohemagglutinin-P