C/EBP transcription factors mediate epicardial activation during heart development and injury

Science. 2012 Dec 21;338(6114):1599-603. doi: 10.1126/science.1229765. Epub 2012 Nov 15.

Abstract

The epicardium encapsulates the heart and functions as a source of multipotent progenitor cells and paracrine factors essential for cardiac development and repair. Injury of the adult heart results in reactivation of a developmental gene program in the epicardium, but the transcriptional basis of epicardial gene expression has not been delineated. We established a mouse embryonic heart organ culture and gene expression system that facilitated the identification of epicardial enhancers activated during heart development and injury. Epicardial activation of these enhancers depends on a combinatorial transcriptional code centered on CCAAT/enhancer binding protein (C/EBP) transcription factors. Disruption of C/EBP signaling in the adult epicardium reduced injury-induced neutrophil infiltration and improved cardiac function. These findings reveal a transcriptional basis for epicardial activation and heart injury, providing a platform for enhancing cardiac regeneration.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Aldehyde Oxidoreductases / genetics
  • Aldehyde Oxidoreductases / metabolism
  • Animals
  • Binding Sites
  • CCAAT-Enhancer-Binding Protein-beta / genetics
  • CCAAT-Enhancer-Binding Protein-beta / metabolism
  • CCAAT-Enhancer-Binding Protein-delta / genetics
  • CCAAT-Enhancer-Binding Protein-delta / metabolism
  • CCAAT-Enhancer-Binding Proteins / genetics
  • CCAAT-Enhancer-Binding Proteins / metabolism*
  • Enhancer Elements, Genetic
  • Female
  • Gene Expression Regulation*
  • Gene Expression Regulation, Developmental
  • Heart / embryology
  • Heart / physiopathology*
  • Male
  • Mice
  • Mice, Transgenic
  • Models, Genetic
  • Myocardial Contraction
  • Myocardial Infarction / genetics*
  • Myocardial Infarction / metabolism
  • Myocardial Reperfusion Injury / genetics*
  • Myocardial Reperfusion Injury / metabolism
  • Neutrophil Infiltration
  • Oligonucleotide Array Sequence Analysis
  • Organ Culture Techniques
  • Pericardium / cytology
  • Pericardium / embryology*
  • Pericardium / metabolism*
  • Signal Transduction
  • Uroplakin III / genetics
  • Uroplakin III / metabolism
  • Ventricular Remodeling
  • WT1 Proteins / genetics
  • WT1 Proteins / metabolism

Substances

  • CCAAT-Enhancer-Binding Protein-beta
  • CCAAT-Enhancer-Binding Proteins
  • CEBPA protein, mouse
  • Cebpb protein, mouse
  • Cebpd protein, mouse
  • Upk3b protein, mouse
  • Uroplakin III
  • WT1 Proteins
  • CCAAT-Enhancer-Binding Protein-delta
  • Aldehyde Oxidoreductases
  • RALDH2 protein, mouse

Associated data

  • GEO/GSE41959