Novel carbocyclic nucleoside analogs suppress glomerular mesangial cells proliferation and matrix protein accumulation through ROS-dependent mechanism in the diabetic milieu

Bioorg Med Chem Lett. 2013 Jan 1;23(1):174-8. doi: 10.1016/j.bmcl.2012.10.122. Epub 2012 Nov 7.

Abstract

The synthesis of a series of novel 3,4-cis- and 3,4-trans-substituted carbocyclic nucleoside analogs from protected uracil and thymine is described. The key reaction in the followed synthetic protocols utilized the Mitsunobu reaction to couple 3,4-substituted cyclopentanols to (3)N-benzoyl uracil or (3)N-benzoyl thymine. These molecules were evaluated with regard to their ability to treat diabetic nephropathy. Our results show that two analogs significantly reduced high-glucose induced glomerular mesangial cells proliferation and matrix protein accumulation in vitro and, more interestingly, exhibited an anti-oxidative effect suggesting that the activity may be mediated through ROS-dependent mechanism.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Actins / metabolism
  • Animals
  • Cell Proliferation / drug effects
  • Cells, Cultured
  • Diabetic Neuropathies / metabolism
  • Diabetic Neuropathies / pathology
  • Extracellular Matrix Proteins / metabolism*
  • Fibronectins / metabolism
  • Glomerular Mesangium / cytology
  • Glomerular Mesangium / metabolism
  • Glucose / pharmacology
  • Nucleosides / chemical synthesis
  • Nucleosides / chemistry*
  • Nucleosides / pharmacology
  • Rats
  • Reactive Oxygen Species / metabolism*

Substances

  • Actins
  • Extracellular Matrix Proteins
  • Fibronectins
  • Nucleosides
  • Reactive Oxygen Species
  • Glucose