Marked alteration in phosphorylation of the RB protein during differentiation of human promyelocytic HL60 cells

Oncogene. 1990 Feb;5(2):179-83.

Abstract

The phosphorylation state of the RB protein was found to change markedly during cell differentiation: most of the RB protein was in a highly phosphorylated 115 kD form in human promyelocytic HL60, erythroleukemic K562 and monocytic U937 cells, but during tumor promoter-induced differentiation, the amount of this form decreased and the amount of an under-phosphorylated 110 kD form of the RB protein increased. Induction of differentiation of HL60 cells by dimethylsulfoxide also induced a similar alteration of the RB protein. However, no alteration in the ratio of the two forms was observed in cell lines such as epidermoid carcinoma A431 and adenocarcinoma MKN-7 that were not induced to differentiate by tumor promoter treatment. The highly phosphorylated form and under-phosphorylated form exhibited different affinity for DNA-cellulose and SV40 large T antigen.

MeSH terms

  • Antigens, Polyomavirus Transforming / metabolism
  • Cell Differentiation
  • Humans
  • Leukemia, Promyelocytic, Acute / metabolism*
  • Leukemia, Promyelocytic, Acute / pathology
  • Neoplasm Proteins / metabolism*
  • Phosphorylation
  • Retinoblastoma / genetics*
  • Tetradecanoylphorbol Acetate / pharmacology
  • Tumor Cells, Cultured

Substances

  • Antigens, Polyomavirus Transforming
  • Neoplasm Proteins
  • Tetradecanoylphorbol Acetate