Class III b-tubulin overexpression in gynecologic tumors: implications for the choice of microtubule targeted agents?

Expert Rev Anticancer Ther. 2013 Jan;13(1):63-74. doi: 10.1586/era.12.158.

Abstract

The tubulins are significant players in maintaining microtubule dynamics and have important signaling and apoptotic functions. Alterations in microtubules as a result of changes in tubulin isotype content or polymerization affect the sensitivity of cell lines to tubulin-binding agents (e.g., taxol) in vitro. Epothilones, such as patupilone and ixabepilone, contain a 16-membered macrolide ring and act as competitive inhibitors of taxol. Class III β-tubulin overexpression has been linked to resistance to paclitaxel and correlated with poor survival in ovarian, breast, gastric, non-small-cell lung cancer and unknown primary tumors. Recent data suggest that class III β-tubulin may not only serve as a marker for sensitivity to epothilones, but also as a mediator of a bioaggressive tumor phenotype through activation of multiple cell survival pathways active under stress conditions.

Publication types

  • Review

MeSH terms

  • Antineoplastic Agents / pharmacology
  • Apoptosis / drug effects
  • Drug Resistance, Neoplasm
  • Female
  • Gene Expression Regulation, Neoplastic
  • Genital Neoplasms, Female / drug therapy*
  • Genital Neoplasms, Female / genetics
  • Genital Neoplasms, Female / pathology
  • Humans
  • Microtubules / metabolism
  • Molecular Targeted Therapy
  • Tubulin / genetics*
  • Tubulin Modulators / pharmacology*

Substances

  • Antineoplastic Agents
  • TUBB3 protein, human
  • Tubulin
  • Tubulin Modulators