[Silencing effect of cell-specific RNA interference plasmid pPSMAe/p-shNS-ploy(A) loaded by transgenic vector Tf-PEG-PEI targeting nucleostemin on prostate cancer cells in vitro]

Zhonghua Zhong Liu Za Zhi. 2012 Oct;34(10):725-9. doi: 10.3760/cma.j.issn.0253-3766.2012.10.002.
[Article in Chinese]

Abstract

Objective: To explore the transgenic efficiency of non-viral vector Tf-PEG-PEI and the cell specific silencing effect of plasmid pPSMAe/p-shNS-ploy(A) on prostate cancer cells.

Methods: Polyethyleneimine (PEI) was modified by using polyethylene glycol and transferrin to synthesize the non-viral vector Tf-PEG-PEI. NS-specific plasmids pPSMAe/p-shNS-ploy(A) and Tf-PEG-PEI were used to transfect prostate cancer LNCap and PC-3 cells. The changes of cell morphology, proliferation ability and cell cycle were studied after down-regulating the NS gene level.

Results: Tf-PEG-PEI was successfully modified. After transfection, the PSMA-expressing LNCaP cells became larger and showed more pseudopodia, having a tendency to differentiate. Their cell proliferation ability was reduced, and the detection of cell cycle showed a decrease of S phase and an increase of G(1) phase after knocking down NS gene. These targets were not changed in non-PSMA-expresing PC-3 cells.

Conclusions: The non-viral vector Tf-PEG-PEI has a high ability to transfer targeted gene into target cells. The cellular specificity of short-hairpin RNA transcription driven by PSMAe/p is confirmed by silencing NS gene. The use of cell specific promoter may be an effective strategy of gene therapy for prostate cancer.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Antigens, Surface / genetics*
  • Antigens, Surface / metabolism
  • Cell Cycle
  • Cell Line, Tumor
  • Cell Proliferation*
  • GTP-Binding Proteins / genetics*
  • GTP-Binding Proteins / metabolism
  • Genetic Vectors
  • Glutamate Carboxypeptidase II / genetics*
  • Glutamate Carboxypeptidase II / metabolism
  • Humans
  • Male
  • Nuclear Proteins / genetics*
  • Nuclear Proteins / metabolism
  • Plasmids
  • Polyethylene Glycols
  • Polyethyleneimine / analogs & derivatives
  • Promoter Regions, Genetic
  • Prostatic Neoplasms / pathology*
  • RNA Interference*
  • RNA, Messenger / metabolism
  • RNA, Small Interfering / genetics
  • Transfection
  • Transferrin / genetics

Substances

  • Antigens, Surface
  • GNL3 protein, human
  • Nuclear Proteins
  • RNA, Messenger
  • RNA, Small Interfering
  • Transferrin
  • poly(ethylene glycol)-co-poly(ethyleneimine)
  • Polyethylene Glycols
  • Polyethyleneimine
  • FOLH1 protein, human
  • Glutamate Carboxypeptidase II
  • GTP-Binding Proteins