Association of glutathione S transferases polymorphisms with glaucoma: a meta-analysis

PLoS One. 2013;8(1):e54037. doi: 10.1371/journal.pone.0054037. Epub 2013 Jan 14.

Abstract

Background: Glutathione S transferase (GST) polymorphisms have been considered risk factors for the development of glaucoma, including primary open angle glaucoma (POAG) and other types of glaucoma. However, the results remain controversial. In this study, we have conducted a meta-analysis to assess the association between polymorphisms of GSTM1, GSTT1 and GSTP1 and glaucoma risk.

Methods: Published literature from PubMed and other databases were retrieved. All studies evaluating the association between GSTM1, GSTT1 and GSTP1 polymorphisms and glaucoma risk were included. Pooled odds ratio (OR) and 95% confidence interval (CI) were calculated using random- or fixed-effects model.

Results: Twelve studies on GSTM1 (1109 cases and 844 controls), ten studies on GSTT1 (709 cases and 664 controls) and four studies on GSTP1 (543 cases and 511 controls) were included. By pooling all the studies, either GSTM1 or GSTT1 null polymorphism was not associated with a POAG risk, and this negative association maintained in Caucasian. The GSTP1 Ile 105 Val polymorphism was significantly correlated with increased POAG risk among Caucasian in a recessive model (Val/Val vs. Ile/Ile+Ile/Val: OR, 1.62, 95%CI: 1.00-2.61). Interestingly, increased glaucoma risk was associated with the combined GSTM1 and GSTT1 null genotypes (OR, 2.20; 95% CI, 1.47-3.31), and with the combined GSTM1 null and GSTP1 Val genotypes (OR, 1.86; 95% CI, 1.15-3.01).

Conclusions: This meta-analysis suggests that combinations of GST polymorphisms are associated with glaucoma risk. Given the limited sample size, the associations between single GST polymorphism and glaucoma risk await further investigation.

Publication types

  • Meta-Analysis
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Glaucoma / genetics*
  • Glutathione S-Transferase pi / genetics
  • Glutathione Transferase / genetics*
  • Humans
  • Polymorphism, Genetic / genetics*

Substances

  • glutathione S-transferase T1
  • Glutathione S-Transferase pi
  • Glutathione Transferase
  • glutathione S-transferase M1

Grants and funding

This work was supported by the Zhejiang Key Innovation Team Project of China (No. 2009R50039), the Zhejiang Key Laboratory Fund of China (No. 2011E10006), the National Natural Science Foundation of China (No. 30900273), the Qianjiang Talents Program of Zhejiang Province (2012R10023) and the Fundamental Research Funds for the Central Universities (No. 2011QNA7018, 2012QNA7019). Dr. Guangdi Chen was supported by Technology Foundation for Excellent Overseas Chinese Scholar, Zhejiang Province Human Resources and Social Security Bureau. The funders had no role in study design, data collection and analysis, decision to publish, or preparation of the manuscript.