Interleukin 6 receptor blockade in patients with neuromyelitis optica nonresponsive to anti-CD20 therapy

JAMA Neurol. 2013 Mar 1;70(3):394-7. doi: 10.1001/jamaneurol.2013.1246.

Abstract

Objective: To report first experiences with interleukin 6 receptor inhibition in therapy-resistant neuromyelitis optica (NMO).

Design: Retrospective case series.

Setting: Neurology department at a tertiary referral center.

Patients: Patients with an aggressive course of NMO switched to tocilizumab after failure of anti-CD20 therapy.

Main outcome measures: Annualized relapse rate and disability progression measured by the Expanded Disability Status Scale.

Results: We report 3 female patients with a median age of 39 years (range, 26-40 years) and aquaporin 4-positive NMO. All patients had been treated with different immunosuppressive and immunomodulating agents, followed by 1 to 3 cycles of rituximab. Despite complete CD20-cell depletion during rituximab therapy, the median annualized relapse rate was 3.0 (range, 2.3-3.0) and the median Expanded Disability Status Scale score increased from 5.0 (range, 4.5-7.0) to 6.5 (range, 5.0-7.0). After the switch to tocilizumab (median duration of therapy, 18 months), the median annualized relapse rate decreased to 0.6 (range, 0-1.3). A total of 2 relapses occurred; however, they were mild and there were no changes in clinical disability.

Conclusions: Interleukin 6 receptor-blocking therapy can be effective in therapy-resistant cases of NMO. Larger controlled studies are needed to confirm the efficacy of tocilizumab.

Publication types

  • Case Reports
  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • Adult
  • Antibodies, Monoclonal, Murine-Derived / therapeutic use
  • Antigens, CD20 / immunology*
  • Female
  • Humans
  • Neuromyelitis Optica / diagnosis
  • Neuromyelitis Optica / drug therapy*
  • Neuromyelitis Optica / immunology*
  • Receptors, Interleukin-6 / antagonists & inhibitors*
  • Receptors, Interleukin-6 / physiology*
  • Retrospective Studies
  • Rituximab
  • Treatment Outcome

Substances

  • Antibodies, Monoclonal, Murine-Derived
  • Antigens, CD20
  • Receptors, Interleukin-6
  • Rituximab