Quantitative NMR analysis of Erk activity and inhibition by U0126 in a panel of patient-derived colorectal cancer cell lines

Biochim Biophys Acta. 2013 Jul;1834(7):1396-401. doi: 10.1016/j.bbapap.2013.01.023. Epub 2013 Jan 27.

Abstract

We comparatively analyzed the basal activity of extra-cellular signal-regulated kinase (Erk1/2) in lysates of 10 human colorectal cancer cell lines by semi-quantitative Western blotting and time-resolved NMR spectroscopy. Both methods revealed heterogeneous levels of endogenous Erk1/2 activities in a highly consistent manner. Upon treatment with U0126, an inhibitor of mitogen-activated protein kinase kinase (MEK) acting upstream of Erk1/2, Western-blotting and NMR congruently reported specific modulations of cellular phospho-Erk levels that translated into reduced kinase activities. Results obtained in this study highlight the complementary nature of antibody- and NMR-based phospho-detection techniques. They further exemplify the usefulness of time-resolved NMR measurements in providing fast and quantitative readouts of kinase activities and kinase inhibitor efficacies in native cellular environments. This article is part of a Special Issue entitled: Inhibitors of Protein Kinases (2012).

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Biocatalysis / drug effects
  • Blotting, Western
  • Butadienes / pharmacology*
  • Cell Line, Tumor
  • Enzyme Inhibitors / pharmacology
  • HCT116 Cells
  • Humans
  • Kinetics
  • Magnetic Resonance Spectroscopy / methods*
  • Mitogen-Activated Protein Kinase 1 / antagonists & inhibitors*
  • Mitogen-Activated Protein Kinase 1 / metabolism
  • Mitogen-Activated Protein Kinase 3 / antagonists & inhibitors*
  • Mitogen-Activated Protein Kinase 3 / metabolism
  • Nitriles / pharmacology*
  • Phosphorylation / drug effects
  • Reproducibility of Results
  • Substrate Specificity
  • Time Factors

Substances

  • Butadienes
  • Enzyme Inhibitors
  • Nitriles
  • U 0126
  • MAPK1 protein, human
  • Mitogen-Activated Protein Kinase 1
  • Mitogen-Activated Protein Kinase 3