Ultrastructural maturation of human-induced pluripotent stem cell-derived cardiomyocytes in a long-term culture

Circ J. 2013;77(5):1307-14. doi: 10.1253/circj.cj-12-0987. Epub 2013 Feb 9.

Abstract

Background: In the short- to mid-term, cardiomyocytes generated from human-induced pluripotent stem cells (hiPSC-CMs) have been reported to be less mature than those of adult hearts. However, the maturation process in a long-term culture remains unknown.

Methods and results: A hiPSC clone generated from a healthy control was differentiated into CMs through embryoid body (EB) formation. The ultrastructural characteristics and gene expressions of spontaneously contracting EBs were analyzed through 1-year of culture after cardiac differentiation was initiated. The 14-day-old EBs contained a low number of myofibrils, which lacked alignment, and immature high-density Z-bands lacking A-, H-, I-, and M-bands. Through the long-term culture up to 180 days, the myofibrils became more tightly packed and formed parallel arrays accompanied by the appearance of mature Z-, A-, H-, and I-bands, but not M-bands. Notably, M-bands were finally detected in 360-day-old EBs. The expression levels of the M-band-specific genes in hiPSC-CMs remained lower in comparison with those in the adult heart. Immunocytochemistry indicated increasing number of MLC2v-positive/MLC2a-negative cells with decreasing number of MLC2v/MLC2a double-positive cells, indicating maturing of ventricular-type CMs.

Conclusions: The structural maturation process of hiPSC-CMs through 1-year of culture revealed ultrastructural sarcomeric changes accompanied by delayed formation of M-bands. Our study provides new insight into the maturation process of hiPSC-CMs.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Biomarkers / metabolism
  • Cell Differentiation*
  • Cell Line
  • Cell Lineage*
  • Gene Expression Regulation, Developmental
  • Humans
  • Immunohistochemistry
  • Induced Pluripotent Stem Cells / metabolism
  • Induced Pluripotent Stem Cells / ultrastructure*
  • Microscopy, Electron, Transmission
  • Muscle Proteins / genetics
  • Muscle Proteins / metabolism
  • Myocardial Contraction
  • Myocytes, Cardiac / metabolism
  • Myocytes, Cardiac / ultrastructure*
  • Sarcomeres / diagnostic imaging*
  • Sarcomeres / metabolism
  • Time Factors
  • Ultrasonography

Substances

  • Biomarkers
  • Muscle Proteins