Abstract
A structure-based computational approach was used to rationally design peptide inhibitors that can target an E3 ligase (SCF(Fbx4) )-substrate (TRF1) interface and subsequent ubiquitylation. Characterization of the inhibitors demonstrates that our sequence-optimization protocol results in an increase in peptide-TRF1 affinity without compromising peptide-protein specificity.
Copyright © 2013 WILEY-VCH Verlag GmbH & Co. KGaA, Weinheim.
Publication types
-
Research Support, N.I.H., Extramural
-
Research Support, U.S. Gov't, Non-P.H.S.
MeSH terms
-
Amino Acid Sequence
-
Drug Design
-
F-Box Proteins / antagonists & inhibitors*
-
F-Box Proteins / chemistry
-
F-Box Proteins / metabolism*
-
Humans
-
Models, Molecular
-
Molecular Sequence Data
-
Mutation
-
Peptides / chemistry*
-
Peptides / genetics
-
Peptides / pharmacology*
-
Protein Binding / drug effects*
-
Protein Interaction Maps / drug effects
-
Telomeric Repeat Binding Protein 1 / antagonists & inhibitors
-
Telomeric Repeat Binding Protein 1 / chemistry
-
Telomeric Repeat Binding Protein 1 / metabolism
-
Ubiquitination / drug effects
Substances
-
F-Box Proteins
-
FBXO4 protein, human
-
Peptides
-
Telomeric Repeat Binding Protein 1