The liver X receptor pathway is highly upregulated in rheumatoid arthritis synovial macrophages and potentiates TLR-driven cytokine release

Ann Rheum Dis. 2013 Dec;72(12):2024-31. doi: 10.1136/annrheumdis-2012-202872. Epub 2013 Feb 23.

Abstract

Objectives: Macrophages are central to the inflammatory processes driving rheumatoid arthritis (RA) synovitis. The molecular pathways that are induced in synovial macrophages and thereby promote RA disease pathology remain poorly understood.

Methods: We used microarray to characterise the transcriptome of synovial fluid (SF) macrophages compared with matched peripheral blood monocytes from patients with RA (n=8).

Results: Using in silico pathway mapping, we found that pathways downstream of the cholesterol activated liver X receptors (LXRs) and those associated with Toll-like receptor (TLR) signalling were upregulated in SF macrophages. Macrophage differentiation and tumour necrosis factor α promoted the expression of LXRα. Furthermore, in functional studies we demonstrated that activation of LXRs significantly augmented TLR-driven cytokine and chemokine secretion.

Conclusions: The LXR pathway is the most upregulated pathway in RA synovial macrophages and activation of LXRs by ligands present within SF augments TLR-driven cytokine secretion. Since the natural agonists of LXRs arise from cholesterol metabolism, this provides a novel mechanism that can promote RA synovitis.

Keywords: Cytokines; Inflammation; Rheumatoid Arthritis; Synovial Fluid.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Aged
  • Arthritis, Rheumatoid / genetics
  • Arthritis, Rheumatoid / metabolism*
  • Arthritis, Rheumatoid / pathology
  • Cell Differentiation / physiology
  • Cytokines / metabolism
  • Female
  • Gene Expression Profiling / methods
  • Humans
  • Liver X Receptors
  • Macrophages / drug effects
  • Macrophages / metabolism*
  • Macrophages / pathology
  • Male
  • Middle Aged
  • Monocytes / metabolism
  • Oligonucleotide Array Sequence Analysis / methods
  • Orphan Nuclear Receptors / biosynthesis*
  • Orphan Nuclear Receptors / genetics
  • Orphan Nuclear Receptors / physiology
  • Signal Transduction / physiology
  • Synovial Fluid / metabolism*
  • Synovitis / metabolism
  • Toll-Like Receptors / physiology*
  • Tumor Necrosis Factor-alpha / pharmacology
  • Up-Regulation

Substances

  • Cytokines
  • Liver X Receptors
  • NR1H3 protein, human
  • Orphan Nuclear Receptors
  • Toll-Like Receptors
  • Tumor Necrosis Factor-alpha