To target or not to target APRIL in systemic lupus erythematosus: that is the question!

Arthritis Res Ther. 2013 Feb 25;15(1):107. doi: 10.1186/ar4160.

Abstract

Among the cytokines that regulate B-cell homeostasis are the TNF-like ligands B-lymphocyte stimulator (BLyS; also B-cell activation factor) and a proliferation-inducing ligand (APRIL). BLyS and APRIL share two receptors; that is, B-cell maturation antigen and transmembrane activator and CAML interactor. Therapeutic approaches using biologics are limited for treatment of lupus patients. One previously approved drug is belimumab, which antagonizes the B-cell stimulator BLyS. Atacicept, another biologic inhibiting BLyS and APRIL, was terminated for serious adverse events--raising the question of whether APRIL should be neutralized in autoimmune diseases.

Publication types

  • Editorial
  • Comment

MeSH terms

  • Biomarkers / blood*
  • Female
  • Humans
  • Kidney / metabolism*
  • Lupus Nephritis / blood*
  • Male
  • Tumor Necrosis Factor Ligand Superfamily Member 13 / blood*

Substances

  • Biomarkers
  • TNFSF13 protein, human
  • Tumor Necrosis Factor Ligand Superfamily Member 13