Probucol inhibits hydrogen peroxide to induce apoptosis of vascular smooth muscle cells

Mol Med Rep. 2013 Apr;7(4):1185-90. doi: 10.3892/mmr.2013.1299. Epub 2013 Jan 29.

Abstract

The aim of this study was to investigate the effect of the antioxidant probucol on hydrogen peroxide (H2O2)-induced vascular smooth muscle cell (VSMC) apoptosis. H2O2 (1 mmol/l) was used as the VSMC apoptosis-inducing agent, and 100, 10 and 1 µmol/l probucol concentrations were incubated with cells for 6 h. The cell apoptosis was observed using Annexin V-FITC, TUNEL and Hoechst 33258 staining methods. H2O2 promoted VSMC apoptosis, ASK-1 protein expression increased and Trx-l protein expression decreased. Probucol reduced H2O2-induced apoptosis in a dose-dependent manner; while ASK-1 protein expression decreased, Trx-1 protein expression increased. Probuco1 is capable of antagonizing the H2O2-induced apoptosis in VSMCs, which may be related to the decrease in ASK-1 protein expression and the increase in Trx-1 protein expression.

MeSH terms

  • Animals
  • Antioxidants / pharmacology*
  • Apoptosis / drug effects
  • Cell Cycle Proteins / biosynthesis*
  • Cell Line
  • Gene Expression Regulation
  • Hydrogen Peroxide / pharmacology
  • MAP Kinase Kinase Kinase 5 / biosynthesis*
  • Muscle, Smooth, Vascular / drug effects
  • Muscle, Smooth, Vascular / growth & development
  • Probucol / pharmacology*
  • Rats
  • Thioredoxins / biosynthesis*

Substances

  • Antioxidants
  • Cell Cycle Proteins
  • Thioredoxins
  • Hydrogen Peroxide
  • MAP Kinase Kinase Kinase 5
  • MAP3K5 protein, human
  • Probucol