Inflammatory markers in the second trimester prior to clinical onset of preeclampsia, intrauterine growth restriction, and spontaneous preterm birth

Inflammation. 2013 Aug;36(4):907-13. doi: 10.1007/s10753-013-9619-x.

Abstract

Low-grade inflammation has been associated with pregnancy complications including preeclampsia (PE), intrauterine growth restriction (IUGR), and spontaneous preterm birth (SPB). In an unmatched, nested case-control study, we assessed the possible predictive association of maternal C-reactive protein (CRP), interferon-γ-inducible protein 10 (IP-10), and soluble urokinase plasminogen activator receptor (suPAR) in second trimester plasma samples in relation to later development of PE (n = 29), IUGR (n = 53), and SPB (n = 9). Inflammatory marker levels in these groups were compared to normotensive healthy pregnant controls (n = 127). We found no statistically significant difference in CRP, IP-10, or suPAR in second trimester plasma samples from pregnant women with later PE, IUGR, and SPB when compared to normotensive healthy controls. Second trimester plasma samples of CRP, IP-10, and suPAR cannot be used as a prognostic marker for PE, IUGR, and SPB.

MeSH terms

  • Adolescent
  • Adult
  • Biomarkers / blood*
  • C-Reactive Protein / analysis
  • Case-Control Studies
  • Chemokine CXCL10 / blood
  • Female
  • Fetal Growth Retardation / diagnosis*
  • Humans
  • Inflammation / blood
  • Pre-Eclampsia / diagnosis*
  • Pregnancy
  • Pregnancy Trimester, Second / blood
  • Premature Birth / diagnosis*
  • Receptors, Urokinase Plasminogen Activator / blood
  • Young Adult

Substances

  • Biomarkers
  • CXCL10 protein, human
  • Chemokine CXCL10
  • PLAUR protein, human
  • Receptors, Urokinase Plasminogen Activator
  • C-Reactive Protein