Deletion of miRNAs in bone marrow prevents streptozotocin-induced murine autoimmune diabetes but deletion of miR-155 does not

Cell Cycle. 2013 Apr 1;12(7):1151-2. doi: 10.4161/cc.24365. Epub 2013 Mar 25.
No abstract available

Keywords: Dicer; knockout mouse; miRNAs; mir-155; streptozotocin; type 1 diabetes.

Publication types

  • Letter
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Bone Marrow / metabolism*
  • DEAD-box RNA Helicases / antagonists & inhibitors
  • DEAD-box RNA Helicases / genetics
  • DEAD-box RNA Helicases / metabolism
  • Diabetes Mellitus, Experimental / genetics
  • Diabetes Mellitus, Experimental / metabolism
  • Diabetes Mellitus, Experimental / prevention & control*
  • Killer Cells, Natural / cytology
  • Killer Cells, Natural / immunology
  • Mice
  • Mice, Knockout
  • MicroRNAs / antagonists & inhibitors
  • MicroRNAs / metabolism*
  • Ribonuclease III / antagonists & inhibitors
  • Ribonuclease III / genetics
  • Ribonuclease III / metabolism
  • T-Lymphocytes, Regulatory / immunology
  • T-Lymphocytes, Regulatory / metabolism

Substances

  • MicroRNAs
  • Mirn155 microRNA, mouse
  • Dicer1 protein, mouse
  • Ribonuclease III
  • DEAD-box RNA Helicases