NR5A1 (SF-1) mutations are not a major cause of primary ovarian insufficiency

J Clin Endocrinol Metab. 2013 May;98(5):E1017-21. doi: 10.1210/jc.2012-4111. Epub 2013 Mar 29.

Abstract

Context: Primary ovarian insufficiency (POI) is a disorder affecting approximately 1% of women under the age of 40 years. NR5A1 (SF-1) mutations have been recently reported in association with POI.

Objective: Our objective was to evaluate the frequency and functional impact of NR5A1 variants in POI.

Patients and methods: One hundred eighty patients diagnosed with idiopathic POI were screened for NR5A1 mutations and functional analysis was performed for the identified variants. The DNA-binding capacity of the variants was evaluated by means of EMSA, while their transcriptional activity was assessed using luciferase reporter assays.

Results: Sequencing the NR5A1 gene revealed 4 missense variants in 3 patients. These patients were aged 20, 25, and 33 years at diagnosis and presented with secondary amenorrhea. None of them presented a syndromic form, although 2 had a familial history of POI. The functional analysis carried out for these missense variants showed no significant difference in DNA binding capacity or in transcriptional activity compared to wild-type NR5A1.

Conclusions: Our study in a large cohort of patients with POI showed the prevalence of NR5A1 mutations to be low (1.6%, upper 95% confidence interval 3.5%). Moreover, no functional impact was observed. Overall, in contrast with the initial report, our results exclude NR5A1 mutations as a major genetic cause of POI.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Africa, Northern
  • Amino Acid Sequence
  • Amino Acid Substitution
  • Cohort Studies
  • Europe
  • Family Health
  • Female
  • Genes, Reporter
  • Genetic Association Studies
  • Genetic Predisposition to Disease*
  • Humans
  • Molecular Sequence Data
  • Mutation, Missense*
  • Primary Ovarian Insufficiency / genetics*
  • Primary Ovarian Insufficiency / metabolism
  • Primary Ovarian Insufficiency / physiopathology
  • Recombinant Proteins / metabolism
  • Reproducibility of Results
  • Sequence Alignment
  • Steroidogenic Factor 1 / chemistry
  • Steroidogenic Factor 1 / genetics*
  • Steroidogenic Factor 1 / metabolism
  • Young Adult

Substances

  • NR5A1 protein, human
  • Recombinant Proteins
  • Steroidogenic Factor 1