A study on the polymorphisms of the renin-angiotensin system pathway genes for their effect on blood pressure levels in males from Algeria

J Renin Angiotensin Aldosterone Syst. 2014 Mar;15(1):1-6. doi: 10.1177/1470320313485898. Epub 2013 Apr 16.

Abstract

Introduction: Several studies have assessed the relationship between blood pressure (BP) and polymorphisms within the genes encoding angiotensinogen (AGT), angiotensin II type 1 receptor (AT1R) and angiotensin-converting enzyme (ACE). However, considering the relatively large discrepancy in frequency and impact of these variants between ethnic groups and populations, still unavailable data from Algerian population are needed.

Objective: Our purpose is to evaluate the association between the AGT M235T, AT1R +1166A/C and ACE I/D polymorphisms and variations in systolic (SBP), diastolic (DBP) and pulse pressure (PP) values.

Methods: The associations with BP were assessed in a representative sample of 115 male subjects free of coronary heart disease (CHD). The AGT M235T, AT1R +1166A/C and ACE I/D polymorphisms were determined by PCR-ASO and PCR-RFLP analysis, respectively.

Results: We showed no associations between the AGT M235T, AT1R +1166A/C nor the ACE I/D polymorphisms with variations in BP values. However, concerning the ACE I/D polymorphism, subjects carrying the ACE I allele tended to have higher SBP (+4.1 mmHg) and PP values (+3.2 mmHg) than DD subjects (adjusted p = 0.087 and p = 0.102, respectively).

Conclusion: The ACE I/D polymorphism needs further investigation in a larger Algerian study, especially concerning its putative impact on SBP and PP.

Keywords: Algerian population; Angiotensin II type 1 receptor; angiotensin-converting enzyme; angiotensinogen; genetic epidemiology; polymorphisms.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Algeria
  • Angiotensinogen / genetics*
  • Blood Pressure / genetics
  • Blood Pressure / physiology*
  • Gene Frequency
  • Humans
  • Male
  • Middle Aged
  • Peptidyl-Dipeptidase A / genetics*
  • Polymorphism, Genetic*
  • Receptor, Angiotensin, Type 1 / genetics*
  • Renin-Angiotensin System / genetics*

Substances

  • Receptor, Angiotensin, Type 1
  • Angiotensinogen
  • ACE protein, human
  • Peptidyl-Dipeptidase A