[Inhibitory effect of NaV1.9 gene silencing on proliferation, phagocytosis and migration in RAW264.7 cells]

Xi Bao Yu Fen Zi Mian Yi Xue Za Zhi. 2013 Mar;29(3):225-8.
[Article in Chinese]

Abstract

Objective: To establish the cell line with stable voltage-gated sodium channels (VGSCs/NaVs) α subunit NaV1.9 gene silencing through RNA interference (RNAi) in murine RAW264.7 macrophages, and to investigate proliferation, phagocytosis and migration in this cell line.

Methods: The stable NaV1.9-deficient cell line was generated by selection in G418 after the transfection of short hairpin (shRNA) plasmid with Lipofectamine TM2000. RNAi efficiency was qualified by RT-PCR; proliferation ability was measured by CCK-8 assay; cell cycle and phagocytic ability were analyzed by flow cytometry; and migrating ability was detected by Transwell migration assay.

Results: Stable NaV1.9-deficient cell line was established and the expression of NaV1.9 was reduced by 80%. CCK-8 assay and flow cytometry showed that the proliferation of the NaV1.9-deficient cell line was inhibited (P<0.05). Flow cytometry revealed that phagocytic ability was reduced in the cell line (P<0.05). Transwell migration assay demonstrated that migrating ability was depressed in the cell line (P<0.05).

Conclusion: In the stable NaV1.9-deficient cells we successfully constructed, proliferation, phagocytosis and migration were obviously inhibited.

Publication types

  • English Abstract

MeSH terms

  • Animals
  • Cell Cycle / genetics
  • Cell Line
  • Cell Movement / genetics*
  • Cell Proliferation
  • Gene Silencing*
  • Macrophages / cytology*
  • Mice
  • NAV1.9 Voltage-Gated Sodium Channel / deficiency*
  • NAV1.9 Voltage-Gated Sodium Channel / genetics*
  • NAV1.9 Voltage-Gated Sodium Channel / metabolism
  • Phagocytosis / genetics*

Substances

  • NAV1.9 Voltage-Gated Sodium Channel
  • Scn11a protein, mouse