Acute vibration induces transient expression of anabolic genes in the murine intervertebral disc

Arthritis Rheum. 2013 Jul;65(7):1853-64. doi: 10.1002/art.37979.

Abstract

Objective: Low-amplitude whole-body vibration has been adopted for the treatment of back pain and spinal disorders. However, there is limited knowledge of the impact of vibration on the intervertebral disc (IVD). This study was undertaken to examine the effects of acute vibration on anabolic and catabolic pathways in the IVD and to characterize the dependence of these changes on time and frequency.

Methods: Custom-designed platforms were developed to apply acute vibration to ex vivo and in vivo mouse models. Spinal segments (ex vivo) or mice (in vivo) were subjected to vibration (for 30 minutes at 15-90 Hz with peak acceleration at 0.3g), and IVDs were examined at specific time points after vibration. Gene expression was quantified using real-time polymerase chain reaction, and protein levels were examined by quantitative mass spectrometry and immunofluorescence.

Results: In the ex vivo model, acute vibration at 15 Hz induced expression of anabolic genes (aggrecan, biglycan, decorin, type I collagen, and Sox9) and suppressed expression of Mmp13, with the most pronounced changes detected 6 hours following vibration. These beneficial effects were frequency dependent and were no longer evident between 45 and 90 Hz. In vivo, the effects on anabolic gene expression were even more robust and were accompanied by decreased expression of Adamts4, Adamts5, and Mmp3. Moreover, significant increases in the protein levels of aggrecan, biglycan, decorin, and type I collagen were detected in vivo.

Conclusion: These findings demonstrate dramatic anabolic effects of acute vibration on IVD tissue, responses that are dependent on frequency. The similarity of the in vivo and ex vivo responses indicates that at least some effects of vibration are tissue autonomous.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • ADAM Proteins / genetics
  • ADAM Proteins / metabolism
  • ADAMTS4 Protein
  • ADAMTS5 Protein
  • Aggrecans / genetics
  • Aggrecans / metabolism
  • Animals
  • Chondroitin Sulfate Proteoglycans / genetics
  • Chondroitin Sulfate Proteoglycans / metabolism
  • Collagen Type I / genetics
  • Collagen Type I / metabolism
  • Gene Expression Profiling
  • Intervertebral Disc / metabolism*
  • Male
  • Matrix Metalloproteinase 13 / genetics
  • Matrix Metalloproteinase 13 / metabolism
  • Matrix Metalloproteinase 3 / genetics
  • Matrix Metalloproteinase 3 / metabolism
  • Mice
  • Organ Culture Techniques
  • Procollagen N-Endopeptidase / genetics
  • Procollagen N-Endopeptidase / metabolism
  • RNA, Messenger / analysis*
  • SOX9 Transcription Factor / genetics
  • SOX9 Transcription Factor / metabolism
  • Vibration*

Substances

  • Aggrecans
  • Chondroitin Sulfate Proteoglycans
  • Collagen Type I
  • RNA, Messenger
  • SOX9 Transcription Factor
  • Sox9 protein, mouse
  • ADAM Proteins
  • ADAMTS5 Protein
  • Adamts5 protein, mouse
  • Matrix Metalloproteinase 13
  • Mmp13 protein, mouse
  • Procollagen N-Endopeptidase
  • Matrix Metalloproteinase 3
  • Mmp3 protein, mouse
  • ADAMTS4 Protein
  • Adamts4 protein, mouse